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Rescue of the abnormal skeletal phenotype in Ts65Dn Down syndrome mice using genetic and therapeutic modulation of trisomic Dyrk1a

July 23, 2015 Amy Clugston

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← Characterizing autism spectrum disorders by key biochemical pathways The Kinase DYRK1A Reciprocally Regulates the Differentiation of Th17 and Regulatory T Cells →

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